Capecitabine, DNA/RNA Synthesis Inhibitor
Por um escritor misterioso
Last updated 20 setembro 2024
Capecitabine is an oral proagent that is converted to its active metabolite, 5-FU, by thymidine phosphorylase. - Mechanism of Action & Protocol.
Capecitabine, C15H22FN3O6
Regulation of thymidylate synthase: an approach to overcome 5-FU resistance in colorectal cancer
is a tumor-selective fluoropyrimidine carbamate, which achieves higher intratumoral 5-FU level with lower toxicity than 5-FU.
Capecitabine
Mechanism of action of 5-fluorouracil: Capecitabine is a prodrug that
5‑Fluorouracil and capecitabine therapies for the treatment of colorectal cancer (Review)
Chemotherapeutic agents - Knowledge @ AMBOSS
POR Clinical benefits of oral capecitabine over intravenous 5-fluorouracyl regimen in case of neoadjuvant chemoradiotherapy followed by surgery for locally advanced rectal cancer
Anti-Cancer Drugs-Medicinal Chemistry
THOC5 complexes with DDX5, DDX17, and CDK12 to regulate R loop structures and transcription elongation rate
Effects of microbiota on anticancer drugs: Current knowledge and potential applications - eBioMedicine
Capecitabine
Capecitabine induces hand-foot syndrome through elevated thymidine phosphorylase-mediated locoregional toxicity and GSDME-driven pyroptosis that can be relieved by tipiracil
Capecitabine
Cellular responses to replication stress: Implications in cancer biology and therapy.
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