Modulation of myeloid and T cells in vivo by Bruton's tyrosine kinase inhibitor ibrutinib in patients with metastatic pancreatic ductal adenocarcinoma
Por um escritor misterioso
Last updated 31 janeiro 2025
Ibrutinib Single-handedly Impacts Immune Environment in Metastatic PDAC
Immunologic Strategies in Pancreatic Cancer: Making Cold Tumors Hot
Protein tyrosine kinase inhibitor resistance in malignant tumors: molecular mechanisms and future perspective
Modulation of myeloid and T cells in vivo by Bruton's tyrosine kinase inhibitor ibrutinib in patients with metastatic pancreatic ductal adenocarcinoma
T cells infiltrate PDA regardless of GM-CSF expression. A-C) IHC of
Frontiers Chimeric Antigen Receptor T-Cells: An Overview of Concepts, Applications, Limitations, and Proposed Solutions
Li Zhang, PhD UCSF Global Cancer Program
Randomized phase II study of the Bruton tyrosine kinase inhibitor acalabrutinib, alone or with pembrolizumab in patients with advanced pancreatic cancer
The recent progress of myeloid‐derived suppressor cell and its targeted therapies in cancers - Ren - 2023 - MedComm - Wiley Online Library
M1 Macrophage-Derived Nanovesicles Potentiate the Anticancer Efficacy of Immune Checkpoint Inhibitors
Scientific rationale for integrative and personalised strategies for pancreatic ductal adenocarcinoma management
WO2016087994A1 - Btk inhibitors to treat solid tumors through modulation of the tumor microenvironment - Google Patents
The reciprocal regulation between host tissue and immune cells in pancreatic ductal adenocarcinoma: new insights and therapeutic implications, Molecular Cancer
Cancers, Free Full-Text
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